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Autism: A Stealth Virus Infection by John Martin, M.D., Ph.D
Introduction
Autism is a diagnostic label applied to neurological disorders of early childhood onset that primarily manifest as
deficits in social interactive skills including speech. Considerable variability exists in the severity of illness in
autistic patients. At one extreme there is a somewhat arbitrary distinction between grossly affected autistic
children and a devastating neurological condition known as childhood disintegration disorder. At the other end
of the spectrum, mildly affected autistic children can share many characteristics with children showing delayed
normal development and/or attention deficit disorders. In recent years there has been a marked increase in
relatively mildly affected autistic children who are commonly referred to as having high functioning autism.
Autism spectrum disorder is usually diagnosed during the second year of life with a failure of normal speech
and impaired socialization. With some children there can be an abrupt loss of certain acquired skills (regressive
autism). In most autistic children, however, subtle indications of impaired and/or delayed development were
present even during the first year of life. Most autistic children will also show a wider variety of clinical
manifestations than implied by a single diagnostic label. These manifestations can include epileptic seizures,
gastrointestinal disorders and endocrine dysfunctions. Recovery from autism can occur especially among
relatively mildly affected children.
While many millions of dollars have been spent on autism-related research, only a few studies have provided
important insights into the underling cause, prevention and treatment of this disease. This report will provide a
brief summary of the issues that are commonly discussed in relation to the possible cause(s) of autism. The
thesis will then be presented that autism is the result of brain damage caused by stealth-adapted viruses.
Accepted Research on Autism
1. Autism has a Genetic Component: This is apparent from the higher incidence of autism among boys. It is
also supported by the relative concordance of disease among monozygotic (genetically identical) twins
compared to dizygotic (genetically different) twins. Still there are examples where only one of two genetically
identical twins has the disease. Several well defined genetic disorders can lead to autistic-like abnormalities,
including Rett's syndrome, an illness that occurs more commonly in girls. Genetic factors typically would not be
expected to change markedly over time and, would not, therefore, easily explain the increasing incidence of
autistic disorders. Nevertheless, since genes regulate many aspects of brain functioning, it is understandable
that genetics will be a contributing factor to the expression of autism from whatever cause. Until the genetics of
brain behavior are far better understood, it is unlikely, however, that gene based therapies will offer real
benefits to presently affected children. .
2. Brain Abnormalities are Present in Autistic Children: Head circumference has been accepted as a
marker of brain size. While essentially normal at birth, the head circumference of many newborn autistic
children increases significantly more than that of normal children. The increase growth rate especially occurs
within the first several months of life and stabilizes thereafter. This important finding indicates that an abnormal
brain developmental process is in place at the time of birth. Brain imaging has also shown enlargements in
certain regions of the brain, most noticeably in parts of the cerebellum. Equally impressive are various
functional studies that show deficits in brain activation upon certain types of sensory stimulation, and in blood
flow patterns in autistic children. The question arises whether these functional changes are a consequence of
autism, rather than its cause. Limited histopathological studies on brain tissue from autistic patients support the
view that certain brain cells are damaged and/or do not normally develop.
Controversial Issues Relating to Autism
1. Autism is the Result of Vaccination. Reports of marked clinical deterioration and even the initial onset of
autism within days of being vaccinated have prompted the view that the vaccine has caused, or has possibly
precipitated, clinical illness. Initially the focus of concern was on diphtheria/petussis/tetanus (DPT) vaccine. The
focus next shifted to MMR (measles/mumps/rubella) vaccine. In the case of measles, vaccine-derived viral
material has been seen using immunohistochemistry and molecular based assays within hyperplastic lymphoid
tissue present in the gastrointestinal tract of autistic children. Similar findings were found in non-autistic children
with lymphoid hyperplasia. Although there are reasons for concerns with live viral vaccines, including the finding
of retroviral related reverse transcriptase activity in MMR vaccines, a primary etiological role of measles vaccine
virus is not supported by data indicating prior brain size abnormality. Proponents of the vaccine theory have
also failed to indicate the lack of cell damage in lymphoid cells positive by immunohistochemistry for measles
antigens. Measles vaccine virus was generally not detectable in the cerebrospinal fluid of autistic children. A
detrimental effect of MMR vaccine has been dismissed by many authorities based on the results of various
epidemiological studies that fail to show a statistically significant correlation between vaccine usage and
disease prevalence.
The focus has since returned to killed vaccines that contain thimerosal as a preservative. It has been argued
that increasing the number of thimerosal containing vaccines has exposed children to neurotoxic levels of
mercury, a component of thimerosal. It has been further suggested that children lacking normal capacity to
either excrete or detoxify mercury may be at particular risk for developing autism. In none of these studies has
scientific validation been obtained for the many suppositions being promulgated. For example, the issue of
impaired excretion of mercury arose from the observation that mercury levels in hair samples obtained from
autistic children were actually lower than those from unaffected children. One could equally well argue that the
autistic children were holding onto mercury for beneficial reasons. Moreover, many autistic children have not
received thimerosal containing vaccines. Environmental mercury contamination is a feature of gold mining
operations in developing countries that have not experienced an unusually high incidence of autism. Mercury
was a common component in 19th century medicines and when poisoning was reported, kidney rather than
brain damage tended to dominate the clinical picture. Unfortunately, emotional rather than scientific arguments
have been thrust onto parents with a disregard for contradictory and non-supportive data.
2. Autism is a Primary Biochemical Disorder. Elevated levels of one or more neuropeptides were reported
to be present in neonatal blood samples obtained from virtually all children subsequently diagnosed with
autism. Although these observations were not confirmed in a subsequent study using a different methodology,
the inference is again supportive of brain damage being present prior to birth. Elevated serum levels of the
neuropeptide BDNF, have also been reported in autistic children. This is interpreted as a possible response to
ongoing brain damage in autistic children. Urine analyses on autistic children have also shown markedly
elevated levels of various metabolic products. An inference is that the levels of these chemicals would also be
increased in the brain and that they somehow interfere with normal brain function. A prime example is the
opiate-like peptides resulting from incomplete digestive breakdown of casein and gluten proteins. This view is
bolstered by noticeable clinical improvements in some autistic children when placed on a casein and gluten free
diet. Peptides from abnormal bacterial and fungal bowel flora may also been suggested as having
neuroregulatory activity whose levels may be reduced by antibiotics. A role for cell associated peptidase is
suggested by unpublished studies that peptidase IV inhibitory fragments may also be present in the urine of
some autistic children.
3. Autism is an Auto-Immune Disease. Antibodies that react with brain tissue or with neuropeptides have
been detected in some autistic children. The idea has been advanced that a brain damaging immune response
has possibly been triggered by a viral illness or by the inoculation of a virus vaccine. These suggestions
disregard the data indicating preexisting brain abnormalities unless it is argued that an auto-immune disease
occurred in the mother.
Autism: A Stealth-Adapted Viral Encephalopathy
The increasing prevalence of autism is consistent with an infectious process. Indeed many commentators have
referred to an autism epidemic. A minority of cases of autism result from prenatal infection with conventional
viruses, including human cytomegalovirus. It is not unreasonable, therefore, to suspect that many other cases
of autism may also be the result of atypical viral illnesses. Viral infection could render a person susceptible to
further brain damage from vaccines and other environmental factors. Several lines of evidence exist that are
supportive of, or at least consistent with a stealth virus cause of autism. These data provide a framework for
therapy and management of autistic children.
1. Blood samples from autistic children consistently induce readily identifiable cell damaging (cytopathic) effects
using viral culture methods adapted for the detection of stealth adapted viruses. Stealth virus testing of blood
samples from autistic children has been subjected to formal "double blind" analyses. Furthermore,
cerebrospinal fluid and gastrointestinal biopsy have similarly yielded positive cultures. Clinical testing for stealth
adapted viruses was discontinued in 2002 under orders from the Federal Government.
2. Assays intended to detect conventional viruses and bacterial pathogens are sometimes positive in autistic
children and their parents. These results can be attributed to serological and/or molecular cross-reactivity with
stealth virus components. For example, various bacteria-derived genes have been detected in cultures of
stealth-adapted viruses that can explain misdiagnoses of Lyme disease or mycoplasma infections.
3. Direct studies have provided evidence for stealth virus infections among family members of autistic children.
Many of the family members will, upon close questioning, report symptoms of neurological and/or immunological
dysfunction consistent with an underlying pervasive infectious disease.
4. The cytopathic effect caused by stealth adapted viruses is enhanced by including live measles vaccine virus
in the cultures, or by the addition of sub-lethal levels of toxins such as thimerosal. A reasonable argument can
be advanced that stealth adapted virus positive infants should be exempted from the overt immunological
stimulation induced by repeated inoculations of mixed vaccines, whether live or containing thimerosal. The
focus of Public Health concern should, however, be more on stealth adapted viruses than potential precipitating
factors.
5. Knowing that a newborn infant is infected with a stealth adapted virus would encourage efforts to develop
and to sustain strong interpersonal behaviors as he or she undertakes the difficult tasks of acquiring language
and emotional relationship skills.
6. Most importantly, studies on stealth adapted viruses have led to the recognition of an alternative
(non-mitochondria) mechanism by which cells can obtain energy. Cultures of stealth adapted viruses undergo a
healing process mediated by materials termed alternative cellular energy pigments (ACE pigments). Various
natural products have ACE pigment activity. Although marketable as dietary supplements, they can not legally
be promoted for disease therapy. It is even difficult to obtain FDA approval for investigational studies on some
of these products. Enhancing the ACE pathway has, nevertheless, helped expedite healing from conventional
viruses, such as HSV and HPV. In another study, an ACE product markedly alleviated diarrhea in children living
in a developing country.
Three issues appear to be limiting the acceptance of stealth adapted virus infection as the underlying cause of
autism. Sadly, one of these factors is the money being made out of the autism epidemic. Parents pay to
attended meetings in which they are presented with scientifically incoherent and commonly contradictory
presentations by so called experts. In some of these meetings, eager parents need to pay more for a private
session with a speaker as if really important information is being withheld from the public talk. More
disconcerting is to see a speaker, fresh from receiving a standing ovation, devising with fellow physicians and
healthcare providers a franchising opportunity to profit from the calamity that has befallen families with autistic
children. Equally bad are discussions of kickback schemes to encourage ordering of unwarranted laboratory
tests, or the peddling of unproven medical devices as income generating additions to questionable medical
practice. Biased opinions are also encouraged by being given monetary value as lawyers solicit support for
planned litigation against vaccine manufacturers.
The second major factor is that Public Health authorities are reluctant to embrace the concept of a viral
epidemic, especially one that could be traced in part to vaccine contamination. This attitude was probably
largely responsible for prohibiting further clinical testing for stealth adapted viruses. In spite of data indicating
the presence of monkey cytomegalovirus DNA in licensed polio virus vaccines, neither CDC, NIH or FDA have
publicly tested blood samples from autistic children for the presence of stealth adapted viruses.
The third factor is that most of those involved in autism research and clinical care are simply not well trained
even in conventional virology yet alone fully understand the concept of stealth adaptation. Stealth adapted
viruses do not provoke an inflammatory reaction, the accepted hallmark of an infectious disease; and can be
somewhat difficult to culture. New concepts are often resisted but especially so if they undermine a profitable
enterprise. Founders and directors of many autism support groups have seemingly been overly influenced by
those making their living from autistic children. In spite of millions of dollars expenditures, the autism epidemic
continues.
I firmly believe that parents have the right to request/demand that Federal and/or State Health Departments do
appropriate virus cultures on a sampling of autistic children. The methods need to be appropriate for the
detection of stealth adapted viruses and have been published. The California Department of Health has also
been provided very detailed protocols. The Government has access to several isolates of stealth adapted
viruses deposited with the American Type Culture Collection, a national repository of biological samples. The
Centers for Diseases Control and Prevention (CDC) can also contact individuals infected with monkey derived
stealth adapted viruses that presumably originally entered humans from contaminated polio virus vaccines.
This is a call for effective action; do what it takes to get the cultures done.
Confirmation of a stealth virus cause of autism will hopefully be followed by the demonstration of clinical
improvements in children treated with anti-stealth virus therapies. Although the immune system is ineffective,
the body has an auxiliary defense system based on generating an alternative (non-mitochondria) pathway of
cellular energy. Various products are becoming available that can provide alternative cellular energy (ACE)
and/or activate this pathway. Clinical trials using these methods are urgently required.
Date: 3/9/06
Author: W. John Martin, M.D., Ph.D.
Source: Institute of Progressive Medicine
If anyone would like to e-mail me directly, please do so at s3support@mail.com I can also host a
teleconference if enough parents are interested. I would appreciate receiving copies of any correspondence
with CDC, State Health Departments or politicians. The web site www.s3support.com has additional information
on stealth adapted viruses and the ACE pathway.
Autism is a diagnostic label applied to neurological disorders of early childhood onset that primarily manifest as
deficits in social interactive skills including speech. Considerable variability exists in the severity of illness in
autistic patients. At one extreme there is a somewhat arbitrary distinction between grossly affected autistic
children and a devastating neurological condition known as childhood disintegration disorder. At the other end
of the spectrum, mildly affected autistic children can share many characteristics with children showing delayed
normal development and/or attention deficit disorders. In recent years there has been a marked increase in
relatively mildly affected autistic children who are commonly referred to as having high functioning autism.
Autism spectrum disorder is usually diagnosed during the second year of life with a failure of normal speech
and impaired socialization. With some children there can be an abrupt loss of certain acquired skills (regressive
autism). In most autistic children, however, subtle indications of impaired and/or delayed development were
present even during the first year of life. Most autistic children will also show a wider variety of clinical
manifestations than implied by a single diagnostic label. These manifestations can include epileptic seizures,
gastrointestinal disorders and endocrine dysfunctions. Recovery from autism can occur especially among
relatively mildly affected children.
While many millions of dollars have been spent on autism-related research, only a few studies have provided
important insights into the underling cause, prevention and treatment of this disease. This report will provide a
brief summary of the issues that are commonly discussed in relation to the possible cause(s) of autism. The
thesis will then be presented that autism is the result of brain damage caused by stealth-adapted viruses.
Accepted Research on Autism
1. Autism has a Genetic Component: This is apparent from the higher incidence of autism among boys. It is
also supported by the relative concordance of disease among monozygotic (genetically identical) twins
compared to dizygotic (genetically different) twins. Still there are examples where only one of two genetically
identical twins has the disease. Several well defined genetic disorders can lead to autistic-like abnormalities,
including Rett's syndrome, an illness that occurs more commonly in girls. Genetic factors typically would not be
expected to change markedly over time and, would not, therefore, easily explain the increasing incidence of
autistic disorders. Nevertheless, since genes regulate many aspects of brain functioning, it is understandable
that genetics will be a contributing factor to the expression of autism from whatever cause. Until the genetics of
brain behavior are far better understood, it is unlikely, however, that gene based therapies will offer real
benefits to presently affected children. .
2. Brain Abnormalities are Present in Autistic Children: Head circumference has been accepted as a
marker of brain size. While essentially normal at birth, the head circumference of many newborn autistic
children increases significantly more than that of normal children. The increase growth rate especially occurs
within the first several months of life and stabilizes thereafter. This important finding indicates that an abnormal
brain developmental process is in place at the time of birth. Brain imaging has also shown enlargements in
certain regions of the brain, most noticeably in parts of the cerebellum. Equally impressive are various
functional studies that show deficits in brain activation upon certain types of sensory stimulation, and in blood
flow patterns in autistic children. The question arises whether these functional changes are a consequence of
autism, rather than its cause. Limited histopathological studies on brain tissue from autistic patients support the
view that certain brain cells are damaged and/or do not normally develop.
Controversial Issues Relating to Autism
1. Autism is the Result of Vaccination. Reports of marked clinical deterioration and even the initial onset of
autism within days of being vaccinated have prompted the view that the vaccine has caused, or has possibly
precipitated, clinical illness. Initially the focus of concern was on diphtheria/petussis/tetanus (DPT) vaccine. The
focus next shifted to MMR (measles/mumps/rubella) vaccine. In the case of measles, vaccine-derived viral
material has been seen using immunohistochemistry and molecular based assays within hyperplastic lymphoid
tissue present in the gastrointestinal tract of autistic children. Similar findings were found in non-autistic children
with lymphoid hyperplasia. Although there are reasons for concerns with live viral vaccines, including the finding
of retroviral related reverse transcriptase activity in MMR vaccines, a primary etiological role of measles vaccine
virus is not supported by data indicating prior brain size abnormality. Proponents of the vaccine theory have
also failed to indicate the lack of cell damage in lymphoid cells positive by immunohistochemistry for measles
antigens. Measles vaccine virus was generally not detectable in the cerebrospinal fluid of autistic children. A
detrimental effect of MMR vaccine has been dismissed by many authorities based on the results of various
epidemiological studies that fail to show a statistically significant correlation between vaccine usage and
disease prevalence.
The focus has since returned to killed vaccines that contain thimerosal as a preservative. It has been argued
that increasing the number of thimerosal containing vaccines has exposed children to neurotoxic levels of
mercury, a component of thimerosal. It has been further suggested that children lacking normal capacity to
either excrete or detoxify mercury may be at particular risk for developing autism. In none of these studies has
scientific validation been obtained for the many suppositions being promulgated. For example, the issue of
impaired excretion of mercury arose from the observation that mercury levels in hair samples obtained from
autistic children were actually lower than those from unaffected children. One could equally well argue that the
autistic children were holding onto mercury for beneficial reasons. Moreover, many autistic children have not
received thimerosal containing vaccines. Environmental mercury contamination is a feature of gold mining
operations in developing countries that have not experienced an unusually high incidence of autism. Mercury
was a common component in 19th century medicines and when poisoning was reported, kidney rather than
brain damage tended to dominate the clinical picture. Unfortunately, emotional rather than scientific arguments
have been thrust onto parents with a disregard for contradictory and non-supportive data.
2. Autism is a Primary Biochemical Disorder. Elevated levels of one or more neuropeptides were reported
to be present in neonatal blood samples obtained from virtually all children subsequently diagnosed with
autism. Although these observations were not confirmed in a subsequent study using a different methodology,
the inference is again supportive of brain damage being present prior to birth. Elevated serum levels of the
neuropeptide BDNF, have also been reported in autistic children. This is interpreted as a possible response to
ongoing brain damage in autistic children. Urine analyses on autistic children have also shown markedly
elevated levels of various metabolic products. An inference is that the levels of these chemicals would also be
increased in the brain and that they somehow interfere with normal brain function. A prime example is the
opiate-like peptides resulting from incomplete digestive breakdown of casein and gluten proteins. This view is
bolstered by noticeable clinical improvements in some autistic children when placed on a casein and gluten free
diet. Peptides from abnormal bacterial and fungal bowel flora may also been suggested as having
neuroregulatory activity whose levels may be reduced by antibiotics. A role for cell associated peptidase is
suggested by unpublished studies that peptidase IV inhibitory fragments may also be present in the urine of
some autistic children.
3. Autism is an Auto-Immune Disease. Antibodies that react with brain tissue or with neuropeptides have
been detected in some autistic children. The idea has been advanced that a brain damaging immune response
has possibly been triggered by a viral illness or by the inoculation of a virus vaccine. These suggestions
disregard the data indicating preexisting brain abnormalities unless it is argued that an auto-immune disease
occurred in the mother.
Autism: A Stealth-Adapted Viral Encephalopathy
The increasing prevalence of autism is consistent with an infectious process. Indeed many commentators have
referred to an autism epidemic. A minority of cases of autism result from prenatal infection with conventional
viruses, including human cytomegalovirus. It is not unreasonable, therefore, to suspect that many other cases
of autism may also be the result of atypical viral illnesses. Viral infection could render a person susceptible to
further brain damage from vaccines and other environmental factors. Several lines of evidence exist that are
supportive of, or at least consistent with a stealth virus cause of autism. These data provide a framework for
therapy and management of autistic children.
1. Blood samples from autistic children consistently induce readily identifiable cell damaging (cytopathic) effects
using viral culture methods adapted for the detection of stealth adapted viruses. Stealth virus testing of blood
samples from autistic children has been subjected to formal "double blind" analyses. Furthermore,
cerebrospinal fluid and gastrointestinal biopsy have similarly yielded positive cultures. Clinical testing for stealth
adapted viruses was discontinued in 2002 under orders from the Federal Government.
2. Assays intended to detect conventional viruses and bacterial pathogens are sometimes positive in autistic
children and their parents. These results can be attributed to serological and/or molecular cross-reactivity with
stealth virus components. For example, various bacteria-derived genes have been detected in cultures of
stealth-adapted viruses that can explain misdiagnoses of Lyme disease or mycoplasma infections.
3. Direct studies have provided evidence for stealth virus infections among family members of autistic children.
Many of the family members will, upon close questioning, report symptoms of neurological and/or immunological
dysfunction consistent with an underlying pervasive infectious disease.
4. The cytopathic effect caused by stealth adapted viruses is enhanced by including live measles vaccine virus
in the cultures, or by the addition of sub-lethal levels of toxins such as thimerosal. A reasonable argument can
be advanced that stealth adapted virus positive infants should be exempted from the overt immunological
stimulation induced by repeated inoculations of mixed vaccines, whether live or containing thimerosal. The
focus of Public Health concern should, however, be more on stealth adapted viruses than potential precipitating
factors.
5. Knowing that a newborn infant is infected with a stealth adapted virus would encourage efforts to develop
and to sustain strong interpersonal behaviors as he or she undertakes the difficult tasks of acquiring language
and emotional relationship skills.
6. Most importantly, studies on stealth adapted viruses have led to the recognition of an alternative
(non-mitochondria) mechanism by which cells can obtain energy. Cultures of stealth adapted viruses undergo a
healing process mediated by materials termed alternative cellular energy pigments (ACE pigments). Various
natural products have ACE pigment activity. Although marketable as dietary supplements, they can not legally
be promoted for disease therapy. It is even difficult to obtain FDA approval for investigational studies on some
of these products. Enhancing the ACE pathway has, nevertheless, helped expedite healing from conventional
viruses, such as HSV and HPV. In another study, an ACE product markedly alleviated diarrhea in children living
in a developing country.
Three issues appear to be limiting the acceptance of stealth adapted virus infection as the underlying cause of
autism. Sadly, one of these factors is the money being made out of the autism epidemic. Parents pay to
attended meetings in which they are presented with scientifically incoherent and commonly contradictory
presentations by so called experts. In some of these meetings, eager parents need to pay more for a private
session with a speaker as if really important information is being withheld from the public talk. More
disconcerting is to see a speaker, fresh from receiving a standing ovation, devising with fellow physicians and
healthcare providers a franchising opportunity to profit from the calamity that has befallen families with autistic
children. Equally bad are discussions of kickback schemes to encourage ordering of unwarranted laboratory
tests, or the peddling of unproven medical devices as income generating additions to questionable medical
practice. Biased opinions are also encouraged by being given monetary value as lawyers solicit support for
planned litigation against vaccine manufacturers.
The second major factor is that Public Health authorities are reluctant to embrace the concept of a viral
epidemic, especially one that could be traced in part to vaccine contamination. This attitude was probably
largely responsible for prohibiting further clinical testing for stealth adapted viruses. In spite of data indicating
the presence of monkey cytomegalovirus DNA in licensed polio virus vaccines, neither CDC, NIH or FDA have
publicly tested blood samples from autistic children for the presence of stealth adapted viruses.
The third factor is that most of those involved in autism research and clinical care are simply not well trained
even in conventional virology yet alone fully understand the concept of stealth adaptation. Stealth adapted
viruses do not provoke an inflammatory reaction, the accepted hallmark of an infectious disease; and can be
somewhat difficult to culture. New concepts are often resisted but especially so if they undermine a profitable
enterprise. Founders and directors of many autism support groups have seemingly been overly influenced by
those making their living from autistic children. In spite of millions of dollars expenditures, the autism epidemic
continues.
I firmly believe that parents have the right to request/demand that Federal and/or State Health Departments do
appropriate virus cultures on a sampling of autistic children. The methods need to be appropriate for the
detection of stealth adapted viruses and have been published. The California Department of Health has also
been provided very detailed protocols. The Government has access to several isolates of stealth adapted
viruses deposited with the American Type Culture Collection, a national repository of biological samples. The
Centers for Diseases Control and Prevention (CDC) can also contact individuals infected with monkey derived
stealth adapted viruses that presumably originally entered humans from contaminated polio virus vaccines.
This is a call for effective action; do what it takes to get the cultures done.
Confirmation of a stealth virus cause of autism will hopefully be followed by the demonstration of clinical
improvements in children treated with anti-stealth virus therapies. Although the immune system is ineffective,
the body has an auxiliary defense system based on generating an alternative (non-mitochondria) pathway of
cellular energy. Various products are becoming available that can provide alternative cellular energy (ACE)
and/or activate this pathway. Clinical trials using these methods are urgently required.
Date: 3/9/06
Author: W. John Martin, M.D., Ph.D.
Source: Institute of Progressive Medicine
If anyone would like to e-mail me directly, please do so at s3support@mail.com I can also host a
teleconference if enough parents are interested. I would appreciate receiving copies of any correspondence
with CDC, State Health Departments or politicians. The web site www.s3support.com has additional information
on stealth adapted viruses and the ACE pathway.